I assume this means that free floating spike can bind to t-cells via these integrin receptors, correct? What would happen to the t-cells since there isn't a virus to infect them?
Correct, and most of the time the T cell will just quite literally kill itself. But before it will set off a inflammatory chain reaction which is the problem in most cases .
since anything 'covid19' related is connected with CRIME, I find it very important to call NAMES here, in all the scientitic research articles, otherwise the point is gone. I tried to do that in all my posts, starting with the RGD motif in March '22:
I directly link every single source, and give credit when it is due (like Theo being the first on uncovering the integrins pathway), and people can find the name of the authors by clicking the sources.
Recombination would be the only way for it to âmutateâ back, there is a lot of arguments among researchers if this motif is advantageous or not to the virus, but it is clinically significant. Let just say the flexible loops that enable it to do what it does, ainât that flexible anymore (outrageous gross simplification).
How would ivermectin or fenbenazole improve outcomes? Would these interrupt the binding of the spike proteins? What other interventions could improve someone with long covid and people that were foolish enough to get vaxâd? Is it just the FLCCC protocols?
They work by stopping specific inflammatory signals and responses, and no they won't stop the spike from binding to anything, very few molecules will.
I have written about possible interventions on all of that for most of this substack existence. One of them is pinned in the starting page of my substack.
Thanks John, another awesome write up and one that was in my to-do list.
The angiogenesis - cancer link is worthy of follow up.
More on effects on the glycocalyx, which can take months or years to recover:
"Persistent capillary rarefication in long COVID syndrome"
https://doorlesscarp953.substack.com/p/persistent-capillary-rarefication
I assume this means that free floating spike can bind to t-cells via these integrin receptors, correct? What would happen to the t-cells since there isn't a virus to infect them?
Correct, and most of the time the T cell will just quite literally kill itself. But before it will set off a inflammatory chain reaction which is the problem in most cases .
since anything 'covid19' related is connected with CRIME, I find it very important to call NAMES here, in all the scientitic research articles, otherwise the point is gone. I tried to do that in all my posts, starting with the RGD motif in March '22:
https://mejbcart.substack.com/p/dr-bret-weinstein-yuri-deigin-and
and continued more in depth in:
https://mejbcart.substack.com/p/how-much-hiv-is-in-sars-cov-2-faucis
Dr. David Martin openly describes what's at stake here for the involved:
https://forbiddenknowledgetv.net/david-martin-transparently-hiding-again/
The entire covid19 science is paid by taxpaeyrs money, who now, after being genetically modified deserve to know who is behind it all.
I directly link every single source, and give credit when it is due (like Theo being the first on uncovering the integrins pathway), and people can find the name of the authors by clicking the sources.
do you have the link to the first Theo's twitter post about integrins?
No he was banned like 5 times from Twitter đ
Is there any reason to expect that the RGD motif could be selected for in some future variant or does this not confer any survival advantage?
Recombination would be the only way for it to âmutateâ back, there is a lot of arguments among researchers if this motif is advantageous or not to the virus, but it is clinically significant. Let just say the flexible loops that enable it to do what it does, ainât that flexible anymore (outrageous gross simplification).
How would ivermectin or fenbenazole improve outcomes? Would these interrupt the binding of the spike proteins? What other interventions could improve someone with long covid and people that were foolish enough to get vaxâd? Is it just the FLCCC protocols?
They work by stopping specific inflammatory signals and responses, and no they won't stop the spike from binding to anything, very few molecules will.
I have written about possible interventions on all of that for most of this substack existence. One of them is pinned in the starting page of my substack.
And no FLCCC is a rather meh intervention, there are dozens upon dozens of possible interventions, the ones I covered are a fraction of it.