There is another hidden (hehe) aspect of the Latent pathogens aspect of SARS-CoV-2, but I promised a person that I consider a very dear friend that I would not write anything about it. Unless that person sends any sort of signal, I won't.
This is a veiled for of asking permission in case the person reads this.
As you already stated "If anything the Spike is a eugenicist wetdream. The wetest of dreams."
So....it seems (to me) we know what will happen. The "when&how" varies, a little bit from person to person, definitely sooner than expected (with or without peptides)? Correct?
No. I don't believe so, it is just cleaning the gene pool in many ways. If people take care of their health, following at least partially what I have written, they will be mostly fine.
The trick is keeping latent viruses, well, latent, and any infection, minimal.
Great "news". I will keep reminiding myself everytime I feel reluctant to board the intercontinental flights out of concerns about respiratory infections. Thank you.
PS. In Australia, we cant buy peptides (anymore) without :
Well, I try to be truthful, to truth and to my morals. I won't lie, perhaps I should sugar coat it ? As long as you minimize any type of infection, especially respiratory and recover as fast as possible, nothing changes for you, but for many others. Well. Survival of the fittest (actually the most adaptable).
The same is being pushed everywhere in regards to peptides. Medical and pharmaceutical corruption are boundless.
I have 27yo super fit climber that hasn’t been vaccinated but is around a lot of vaccinated people. last summer he got GBS. He was in the hospital for a week and couldn’t walk. Was afraid he would never climb again. Fortunately he is so fit, he basically completely recovered in about a month, with the exception of minor lingering neuropathy on the side of his face that he still has.
Stevens Johnson is frequently simplified to be represented as a drug reaction, but (as some of your articles point out here) in dermatology books there are a whole list of viruses and bacteria that can trigger Stevens Johnson as well as its less severe relative, Erythema Multiform. It's essentially a hyper immune attack that gets launched when cells in the skin present antigens. (Think of it as a visible display of what's probably happening in our hearts, brains, reproductive organs, livers, and blood vessels when those cells have the misfortune of being transfected with spike and presenting those antigens.)
Spike has superantigens lifted from other species built into its sequence: (Dakal TC. Immunobiology. 2021 Sep;226(5):152091. DOI: 10.1016/j.imbio.2021.152091: "..... molecular similarity with 54 antigenic determinants found in twelve pathogenic bacterial species (Mycobacterium tuberculosis, Mycobacterium leprae, Bacillus anthracis, Borrelia burgdorferi, Clostridium perfringens, Clostridium tetani, Helicobacter Pylori, Listeria monocytogenes, Staphylococcus aureus, Streptococcus pyogenes, Vibrio cholera and Yersinia pestis), two malarial parasites (Plasmodium falciparum and Plasmodium knowlesi) and influenza virus A."
Beyond the superantigen sequences, there is also Heat Shock Protein, an old biolab trick, which I find connects the dots between Spike, Stevens Johnson, and GBS. Plus Zika, one of their brilliant previous releases on the world:
Any of these terms sound familiar?: Antibodies against HSP, which may very well develop after feeding the body Spike, are also associated with Graft-Versus-Host-Disease, Stroke, Autoimmune vasculitis, ocular inflammation, sensorineural hearing loss, Meniere’s Disease, Myasthenia Gravis, Lupus, Rheumatoid Arthritis, Asthma, Glomerulo Nephritis, MS, Diabetes I, Schizophrenia, misfolded proteins, neurodegeneration.… Heat shock proteins are also regulators of apoptosis and innate immunity.
Fantastic- explains a lot of what I've heard from others who attribute their conditions to nerves, depression, and more. I want to be sure about one thing though, when you use the term " endotoxin tolerance" what exactly do you mean? I'm understanding it as the body permitting the presence of the various endotoxins of the Sars-cov infection and the vaxx ( spike plus plasmids) and this in fact is basically Sepsis. Please correct me if I'm wrong. Your posts are so informative at a comprehensible level.
Endotoxin tolerance (I will write a entire massive thing on it alone) happens when your cells are exposed to endotoxins, specifically LPS (lipopolysaccharide) the "skin" of some bacteria. Which SARS2 literally manipulates, removes from bacteria and uses for itself.
When exposed to a certain amount (doesn't need to be massive) your immune cells enter that state to avoid a overwhelming inflammatory response (such as cytokine storm). It is a defense mechanism that, as with everything inside us, has a double edge nature. You are protected from a overwhelming response yet becomes susceptible for secondary infections.
I try to cover the science as best I can and make it comprehensible for most people. Sometimes it is really hard because the subjects covered are incredibly complex. Thank you for the kind words though :)
I have a series of questions (leading up to the last one, be patient) I've been thinking about for a couple years now and hoping you have answers.
1. Can sars-cov-2 itself infect cells lacking specific receptors (I think the main one is ACE-2?)
2. Can mRNA using LNP coatings transfect generally all cells within the body, regardless of what receptors they have?
3. Does the spike protein get presented on the surface of cells regardless of how the genetic material was delivered (natural infection vs LNP)? I think a follow on here would be whether the two have different tendencies toward auto-immunity as well.
4. How does all of this work in the case of LNPs introducing mRNA into B and T cells (assuming the answer to #2 is yes, and LNPs do facilitate transfection of these cells)? Does this potentially lead to something different than auto-immunity where I understand the immune system attacks parts distinct from the immune system? Would the immune system essentially commit suicide as it deems itself as foreign?
I've never seen #4 asked or answered anywhere. If it has been addressed previously, please do share so that I can read up.
1. SARS-CoV-2 has a myriad of other receptors, with each one leading to different effects depending on cell type or the protein it binds.
2. LNP was designed to >slice< through any biological membrane, if something binds to a receptor, it activates your immune system or a cellular response, the LNP has no binding, it cuts through... well, everything, to get inside a cell.
3. The spike protein from the virus is attached to the virus, the spike protein produced by translation of mRNA is theoretically presented on the surface of cells but in actuality it often floats around.
4. Antigen Presenting Cells (APC) are responsible for starting a immune response. The autoimmunity is highly complex and I have written extensively about it, it would take at least 20 substacks I wrote to make sense of the autoimmune part and my is just a FRACTION of the complexity of autoimmunity in regards to SARS-CoV-2 and mRNA vaccines.
The autoimmunity is multi-faceted, there is no "singular" causative agent, or mechanism.
So if I understand you correctly, there is nothing "novel" about B and T cells producing spike protein and this can occur naturally. It is also natural for B cells to present antigens on their surfaces, however what is new in the case of mRNA is T cells potentially presenting antigens (spike)? Does this change in T cell behavior potentially lead to other effects such as accumulation of T cells and thickening/clotting in the blood stream?
SARS can't replicate inside Lymphocytes. So no, there is nothing natural in regards to how the mRNA produces the protein regardless of cell type that produces it.
Spike can directly causes endothelial dysfunction by damaging smooth muscle cells, endothelial cells, and directly binds to vWF, among a dozen other ways to induce clotting.
Endotoxins induce clotting, sepsis induces clotting, bacterial infection induces clotting, a bunch of things induced clotting and the Spike can mess with all of them.
The change and B cell and T cell behavior comes from other effects. Presenting antigens to cells is how you elicit a immune response.
The problem is the protein, simple as that. The problem is the Spike.
And the size and chemical structure of the LNPs.
And the shape, structure of the mRNA, and how the body "sees" it. I have addressed all these. I have over 170 (probably 200) pieces written about SARS. It is impossible to say one thing causes X.
Viruses come out of latency from systemic physiological and immunological changes, not just changes in one cell type, which B cells themselves have many subtypes.
PROTOCOLS OF THE MEETINGS OF THE LEARNED ELDERS OF ZION . . . Protocol X – Preparing for Power . . . (((SARS-CoV2)))
❝. . . utterly exhaust humanity with dissention, hatred, struggle, envy and even by the use of torture, by starvation, by the inoculation of diseases. by want, so that the “Goyim” see no other issue than to take refuge in our complete sovereignty in money and in all else.❞
There is another hidden (hehe) aspect of the Latent pathogens aspect of SARS-CoV-2, but I promised a person that I consider a very dear friend that I would not write anything about it. Unless that person sends any sort of signal, I won't.
This is a veiled for of asking permission in case the person reads this.
Dr. SHIVA Ayyadurai, MIT PhD . . . Dr.SHIVA™ INTERVIEW: Why Joe Rogan Fears A Real Scientist . . .
https://www.youtube.com/watch?v=gWt7mQ2-lIo
Thank you for explaining things so clearly.
Baby steps. The real explanation is coming given the sheer complexity of it. First the tryptophan one, than the long COVID/latent viruses one.
As you already stated "If anything the Spike is a eugenicist wetdream. The wetest of dreams."
So....it seems (to me) we know what will happen. The "when&how" varies, a little bit from person to person, definitely sooner than expected (with or without peptides)? Correct?
No. I don't believe so, it is just cleaning the gene pool in many ways. If people take care of their health, following at least partially what I have written, they will be mostly fine.
The trick is keeping latent viruses, well, latent, and any infection, minimal.
Great "news". I will keep reminiding myself everytime I feel reluctant to board the intercontinental flights out of concerns about respiratory infections. Thank you.
PS. In Australia, we cant buy peptides (anymore) without :
1. Comprehensive full blood/endocrine work
2. Specialist Medical Appointment
Total cost around $300 (just for prescreening)
Well, I try to be truthful, to truth and to my morals. I won't lie, perhaps I should sugar coat it ? As long as you minimize any type of infection, especially respiratory and recover as fast as possible, nothing changes for you, but for many others. Well. Survival of the fittest (actually the most adaptable).
The same is being pushed everywhere in regards to peptides. Medical and pharmaceutical corruption are boundless.
Artemisinin and pomegranate peel - can help ferroptosis. Magnesium too.
I have 27yo super fit climber that hasn’t been vaccinated but is around a lot of vaccinated people. last summer he got GBS. He was in the hospital for a week and couldn’t walk. Was afraid he would never climb again. Fortunately he is so fit, he basically completely recovered in about a month, with the exception of minor lingering neuropathy on the side of his face that he still has.
Stevens Johnson is frequently simplified to be represented as a drug reaction, but (as some of your articles point out here) in dermatology books there are a whole list of viruses and bacteria that can trigger Stevens Johnson as well as its less severe relative, Erythema Multiform. It's essentially a hyper immune attack that gets launched when cells in the skin present antigens. (Think of it as a visible display of what's probably happening in our hearts, brains, reproductive organs, livers, and blood vessels when those cells have the misfortune of being transfected with spike and presenting those antigens.)
Spike has superantigens lifted from other species built into its sequence: (Dakal TC. Immunobiology. 2021 Sep;226(5):152091. DOI: 10.1016/j.imbio.2021.152091: "..... molecular similarity with 54 antigenic determinants found in twelve pathogenic bacterial species (Mycobacterium tuberculosis, Mycobacterium leprae, Bacillus anthracis, Borrelia burgdorferi, Clostridium perfringens, Clostridium tetani, Helicobacter Pylori, Listeria monocytogenes, Staphylococcus aureus, Streptococcus pyogenes, Vibrio cholera and Yersinia pestis), two malarial parasites (Plasmodium falciparum and Plasmodium knowlesi) and influenza virus A."
Beyond the superantigen sequences, there is also Heat Shock Protein, an old biolab trick, which I find connects the dots between Spike, Stevens Johnson, and GBS. Plus Zika, one of their brilliant previous releases on the world:
https://medquotes.substack.com/p/bullous-blisters-after-spike?
https://medquotes.substack.com/p/not-new-spike-guillain-barre-heat?
Any of these terms sound familiar?: Antibodies against HSP, which may very well develop after feeding the body Spike, are also associated with Graft-Versus-Host-Disease, Stroke, Autoimmune vasculitis, ocular inflammation, sensorineural hearing loss, Meniere’s Disease, Myasthenia Gravis, Lupus, Rheumatoid Arthritis, Asthma, Glomerulo Nephritis, MS, Diabetes I, Schizophrenia, misfolded proteins, neurodegeneration.… Heat shock proteins are also regulators of apoptosis and innate immunity.
Fantastic- explains a lot of what I've heard from others who attribute their conditions to nerves, depression, and more. I want to be sure about one thing though, when you use the term " endotoxin tolerance" what exactly do you mean? I'm understanding it as the body permitting the presence of the various endotoxins of the Sars-cov infection and the vaxx ( spike plus plasmids) and this in fact is basically Sepsis. Please correct me if I'm wrong. Your posts are so informative at a comprehensible level.
Endotoxin tolerance (I will write a entire massive thing on it alone) happens when your cells are exposed to endotoxins, specifically LPS (lipopolysaccharide) the "skin" of some bacteria. Which SARS2 literally manipulates, removes from bacteria and uses for itself.
When exposed to a certain amount (doesn't need to be massive) your immune cells enter that state to avoid a overwhelming inflammatory response (such as cytokine storm). It is a defense mechanism that, as with everything inside us, has a double edge nature. You are protected from a overwhelming response yet becomes susceptible for secondary infections.
I try to cover the science as best I can and make it comprehensible for most people. Sometimes it is really hard because the subjects covered are incredibly complex. Thank you for the kind words though :)
I have a series of questions (leading up to the last one, be patient) I've been thinking about for a couple years now and hoping you have answers.
1. Can sars-cov-2 itself infect cells lacking specific receptors (I think the main one is ACE-2?)
2. Can mRNA using LNP coatings transfect generally all cells within the body, regardless of what receptors they have?
3. Does the spike protein get presented on the surface of cells regardless of how the genetic material was delivered (natural infection vs LNP)? I think a follow on here would be whether the two have different tendencies toward auto-immunity as well.
4. How does all of this work in the case of LNPs introducing mRNA into B and T cells (assuming the answer to #2 is yes, and LNPs do facilitate transfection of these cells)? Does this potentially lead to something different than auto-immunity where I understand the immune system attacks parts distinct from the immune system? Would the immune system essentially commit suicide as it deems itself as foreign?
I've never seen #4 asked or answered anywhere. If it has been addressed previously, please do share so that I can read up.
1. SARS-CoV-2 has a myriad of other receptors, with each one leading to different effects depending on cell type or the protein it binds.
2. LNP was designed to >slice< through any biological membrane, if something binds to a receptor, it activates your immune system or a cellular response, the LNP has no binding, it cuts through... well, everything, to get inside a cell.
3. The spike protein from the virus is attached to the virus, the spike protein produced by translation of mRNA is theoretically presented on the surface of cells but in actuality it often floats around.
4. Antigen Presenting Cells (APC) are responsible for starting a immune response. The autoimmunity is highly complex and I have written extensively about it, it would take at least 20 substacks I wrote to make sense of the autoimmune part and my is just a FRACTION of the complexity of autoimmunity in regards to SARS-CoV-2 and mRNA vaccines.
The autoimmunity is multi-faceted, there is no "singular" causative agent, or mechanism.
So if I understand you correctly, there is nothing "novel" about B and T cells producing spike protein and this can occur naturally. It is also natural for B cells to present antigens on their surfaces, however what is new in the case of mRNA is T cells potentially presenting antigens (spike)? Does this change in T cell behavior potentially lead to other effects such as accumulation of T cells and thickening/clotting in the blood stream?
SARS can't replicate inside Lymphocytes. So no, there is nothing natural in regards to how the mRNA produces the protein regardless of cell type that produces it.
Spike can directly causes endothelial dysfunction by damaging smooth muscle cells, endothelial cells, and directly binds to vWF, among a dozen other ways to induce clotting.
Endotoxins induce clotting, sepsis induces clotting, bacterial infection induces clotting, a bunch of things induced clotting and the Spike can mess with all of them.
The change and B cell and T cell behavior comes from other effects. Presenting antigens to cells is how you elicit a immune response.
The problem is the protein, simple as that. The problem is the Spike.
And the size and chemical structure of the LNPs.
And the shape, structure of the mRNA, and how the body "sees" it. I have addressed all these. I have over 170 (probably 200) pieces written about SARS. It is impossible to say one thing causes X.
I think these questions may also tie in with your post, in that I think EBV becomes a latent virus, persisting within B cells.
Viruses come out of latency from systemic physiological and immunological changes, not just changes in one cell type, which B cells themselves have many subtypes.
PROTOCOLS OF THE MEETINGS OF THE LEARNED ELDERS OF ZION . . . Protocol X – Preparing for Power . . . (((SARS-CoV2)))
❝. . . utterly exhaust humanity with dissention, hatred, struggle, envy and even by the use of torture, by starvation, by the inoculation of diseases. by want, so that the “Goyim” see no other issue than to take refuge in our complete sovereignty in money and in all else.❞
https://cwspangle.substack.com/p/protocol-x-preparing-for-power-sars