Peru GBS crisis, massive increase in Steve-Johnson Syndrome massive increase
And the hidden things inside...
This will be a mixed piece, parts “science”, parts… opinion ? Criticism ? I have openly criticized “my side” of the whole pandemic debacle for the good part of the last 9 months, and sometimes I am openly hostile to some who misinterpret the evidence or do a pisspoor job researching, engaging in both to effectively use Narrative Warfare (a subfield of Cognitive Warfare).
Narrative warfare can use different techniques to achieve its goal, but the easiest ones to spot are Linguistic manipulation, which involved using deliberate terms to convey a specific message or bring up a emotional response, loaded terms, euphemisms, selective usage of words to convey strong negative connotations are the “tools of the trade”. The second, which could be considered part of linguistic manipulation but should be analyzed on its own is Emotionally loaded language. Emotionally charged words thus shaping narratives can trigger fear, anger, sympathy, or solidarity, among other emotions, which can cloud judgment and lead to biased interpretations. These emotional responses can override critical thinking and rational analysis, making individuals more susceptible to manipulation.
But not entirely negative these moments are since sometimes they lead my (weird) brain into solving pieces of the puzzle. To understand my criticism above at some level, first I will present the piece of news that led me to write this piece, with the second piece of news being as important to the overall proposition.
Earlier today Peru declared a health emergency given the surge in GBS. GBS and SJS are distinct “diseases”, and of course, both are being blamed solely and only on the mRNA vaccines, if you are my reader and keep notes (as I keep hinting for you to do, often) you might have picked a hint of a clue. Stevens-Johnson syndrome (SJS) is effectively classified as a Drug Hypersensitivity reaction, you will often see literature solely focusing on SJS as a drug reaction, and the argument of many is “this is a reaction towards the mRNA”.
In my last Diabetes substack I left a hint, I will leave the quote below.
HHV-6 (one of the pathogens responsible for Long Covid) has been linked as a contributor to T1D. Drug Hypersensitivity Syndrome can be a life-threatening condition, in rare cases leading to Fulminant T1D, and HHV-6 has been implicated as having a role.
Peru experienced an outbreak of GBS at some point in 2019, and studies blamed a bacteria named Campylobacter. Yet…
Association between Guillain-Barré syndrome and Herpes virus family members, A Multiplex PCR study
A Rare Case of Epstein-Barr Virus: Infectious Mononucleosis Complicated by Guillain-Barré Syndrome
The incidence of GBS after an EBV infection was reported to be approximately 10%
Severe Guillain-Barré syndrome following primary infection with varicella zoster virus in an adult
Far from me to overly generalize and outright state this is the only possible explanation, but given all the dynamics of SARS-CoV-2 that I quite extensively wrote about, it clearly points towards a more complex effect than merely “mRNA bad”, an oversimplification that rather annoys me. To be discussed is this another unintended consequence of mRNA+LNP as a platform, the Spike Protein, or everything all at once ?
Since viral reactivation, taking many latent pathogens into an active stage has been tracked and published in many scientific papers, such as these. (I will cite the second paper again soon). Another perspective to keep in mind is that Sepsis (therefore Endotoxic Tolerance) also induces viral reactivation of the same viruses, almost “to a T”.
While I have chosen to abstain from the whole IgG4 trend… well, let me be clear, it wasn’t a trend, it was narrative warfare, I kept researching. And a friend asked “Any IgG4 involvement at all ?”
HHV-6-specific IgG4 was detected in all bone marrow transplant recipients whose HHV-6 IgG antibody titers increased by at least 8-fold. The possibility that IgG4 antibodies may be a marker for HHV-6 reactivation warrants further study.
Of course viral reactivation alone can’t and won’t explain the shift towards IgG4, but it obviously both play a role in some, and it will definitely aid in further shifting and “locking” people into that immune state, given the chronic exposure to antigens.
This was a timely and happy accident, giving me the opportunity to finish and publish my Tryptophan piece and my Long Covid-Viral latency. I have been arguing about viral reactivation and its role in SARS-CoV-2 pathogenicity since 2020, but this was the earliest tweet I could find. Tryptophan one will be published at the earliest tomorrow.
If you chose to support my work in any way, thank you !
There is another hidden (hehe) aspect of the Latent pathogens aspect of SARS-CoV-2, but I promised a person that I consider a very dear friend that I would not write anything about it. Unless that person sends any sort of signal, I won't.
This is a veiled for of asking permission in case the person reads this.
Thank you for explaining things so clearly.