Keep in mind that this experiment (covid) was conducted on a population that is widely vitamin D deficient. Against that backdrop, the final common path of covid and covid vax injuries is harmful inflammation exacerbated by dysfunctional vitamin D deficient immune systems. It’s nice to know how the spike protein is killing us but more important to know how to prevent it…take your vitamin D.
Does this suggest a need to add some dysbiosis mitigation supps to the stack? Probiotics, tryptophan, colustrum right when infected or after jabbed to prevent escalation?
Wow! What amazing research and journalism. We all need to understand what is happening here. The vaccine is designed to immunize us by entering the cell and getting our ribosomes to produce spike protein that provoke an antigenic response. But the spike is itself an antigen that provoked inflammation and thrombosis. Wow! Scary! Set aside the bacteriophage, dysbiosis and reverse transcription. WTF! Monstrous and targeted to the elderly and immune senescence, but hangs around for years, like shingles, waiting to strike. No wonder Xi has zero-Covid policy.
Hello John Paul... This may or may not be something...
I consider this conference abstract from late 2021 to be a critical data point in building a big picture view of what's going on with these jabs. Although other commentators have started to use other data to suggest that everyone getting these shots is sustaining at least some low level of sub-clinical heart damage, the data in this abstract very clearly showed this, and the most elevated signal of all (in the suite of heart attack predictive blood tests called PULS) was Hepatocyte Growth Factor, or HGF. This paper followed the PULS results for 566 patients in a cardiology practice, baselining them before and testing again after double jabbing. The results were astounding, as imputed (i.e. algorithmically determined by the nine or so underlying PULS-specified blood tests) 5-year projected heart attack risk went from a mean of 11% --> 25% post jabs!!! HGF is associated with, i.e. plays a directing role in, T-cell infusion into heart tissue
Anyway, since HGF was among the most elevated of the component tests, I have been researching it over time. It is known to be stimulated by antigens in heart tissue... so it may be as straightforward as spike protein in the heart endothelium... but then I found this;
"To identify physiologic sites of T cell priming in the presence of HGF, we analyzed and compared the production of HGF mRNA by a number of murine tissues, including liver and heart, both in steady-state conditions and 24 hr after the injection of 100 μg LPS. As shown in Figures 3A–3C, in addition to the liver, heart parenchyma was found to express HGF mRNA, particularly in inflammatory settings provoked by endotoxin. Immunohistochemical analysis confirmed that this cytokine is constitutively present in low amounts in cardiac tissue, and it is upregulated by inflammatory stimuli induced by LPS (Figure 3C). In addition, mRNA transcripts for the chemokine CXCL10, but not CCL4 or CCR4 ligands, were found induced in inflamed cardiac tissue (Figure 3D)."
If the wonderful, immune-evading, pseudo-uridine protected spike is transporting LPS... that may be another means by which the heart tissue inflammation cycle kicks off.
Phages as new human viral pathogens a la the Tetz researchers?
"Here, for the first time, we presented the concept of bacteriophages as human pathogens, implying phages as previously overlooked factors that might be associated with human diseases. This concept adds phages and phagobiota to the growing list of factors associated with human health, and moreover suggests that bacteriophages and the alteration of their abundances may be a target for therapeutic intervention. Substantial additional experimental work is required to distinguish which bacteriophages contribute to the development of human diseases and to evaluate the roles of human’s genetic and microbiota susceptibilities therein."
By the way, from March, There is the study, Hijacking time: How Ophiocordyceps fungi could be using ant host clocks to manipulate behavior. https://pubmed.ncbi.nlm.nih.gov/35103986/
Immunological Mechanisms Explaining the Role of Vaccines, IgE, Mast Cells, Histamine, Elevating Ferritin, IL-6, D-dimer, VEGF Levels in COVID-19 and Dengue, Potential Treatments Such as Mast Cell Stabilizers, Antihistamines: Predictions and Confirmations
This is insanity, from my next review - researchers actually used LPS to deliberately induce brain injury:
Wang X, Yu JY, Sun Y, Wang H, Shan H, Wang S. Baicalin protects LPS-induced blood-brain barrier damage and activates Nrf2-mediated antioxidant stress pathway. Int Immunopharmacol. 2021 Jul;96:107725. doi: 10.1016/j.intimp.2021.107725. Epub 2021 May 28. PMID: 34162131.
Dr Isaac Elias researches modified citrus pectin to lower galectin-3. A search of his name will bring up many interviews he has done on subject. His company EcoNugenics sell his version of modified citrus pectin called Pectasol. Just had my galectin levels drawn curious to see what they are had Covid in April.
I’ll look forward to it. I’ve also seen some preliminary data indicating that folic acid disrupts attachment of the spike protein to the furin binding site. So that could be additive to your discussion of natto.
I’m probably way off, but could any of this explain the “extremely unusual” autopsy findings in the congressman’s daughter’s (SADS victim) blood culture? Clostridium sordellii and septicum? Even if they didn’t consider it to be a contributing factor?
Keep in mind that this experiment (covid) was conducted on a population that is widely vitamin D deficient. Against that backdrop, the final common path of covid and covid vax injuries is harmful inflammation exacerbated by dysfunctional vitamin D deficient immune systems. It’s nice to know how the spike protein is killing us but more important to know how to prevent it…take your vitamin D.
Does this suggest a need to add some dysbiosis mitigation supps to the stack? Probiotics, tryptophan, colustrum right when infected or after jabbed to prevent escalation?
Wow! What amazing research and journalism. We all need to understand what is happening here. The vaccine is designed to immunize us by entering the cell and getting our ribosomes to produce spike protein that provoke an antigenic response. But the spike is itself an antigen that provoked inflammation and thrombosis. Wow! Scary! Set aside the bacteriophage, dysbiosis and reverse transcription. WTF! Monstrous and targeted to the elderly and immune senescence, but hangs around for years, like shingles, waiting to strike. No wonder Xi has zero-Covid policy.
Hello John Paul... This may or may not be something...
I consider this conference abstract from late 2021 to be a critical data point in building a big picture view of what's going on with these jabs. Although other commentators have started to use other data to suggest that everyone getting these shots is sustaining at least some low level of sub-clinical heart damage, the data in this abstract very clearly showed this, and the most elevated signal of all (in the suite of heart attack predictive blood tests called PULS) was Hepatocyte Growth Factor, or HGF. This paper followed the PULS results for 566 patients in a cardiology practice, baselining them before and testing again after double jabbing. The results were astounding, as imputed (i.e. algorithmically determined by the nine or so underlying PULS-specified blood tests) 5-year projected heart attack risk went from a mean of 11% --> 25% post jabs!!! HGF is associated with, i.e. plays a directing role in, T-cell infusion into heart tissue
https://www.ahajournals.org/doi/abs/10.1161/circ.144.suppl_1.10712
Anyway, since HGF was among the most elevated of the component tests, I have been researching it over time. It is known to be stimulated by antigens in heart tissue... so it may be as straightforward as spike protein in the heart endothelium... but then I found this;
https://www.cell.com/immunity/fulltext/S1074-7613(15)00212-5
"To identify physiologic sites of T cell priming in the presence of HGF, we analyzed and compared the production of HGF mRNA by a number of murine tissues, including liver and heart, both in steady-state conditions and 24 hr after the injection of 100 μg LPS. As shown in Figures 3A–3C, in addition to the liver, heart parenchyma was found to express HGF mRNA, particularly in inflammatory settings provoked by endotoxin. Immunohistochemical analysis confirmed that this cytokine is constitutively present in low amounts in cardiac tissue, and it is upregulated by inflammatory stimuli induced by LPS (Figure 3C). In addition, mRNA transcripts for the chemokine CXCL10, but not CCL4 or CCR4 ligands, were found induced in inflamed cardiac tissue (Figure 3D)."
If the wonderful, immune-evading, pseudo-uridine protected spike is transporting LPS... that may be another means by which the heart tissue inflammation cycle kicks off.
Thank you so much for the primer (more hard science for dumbo me, YIKES! )
Really, another great post!!!
Pursuant to your assertion that the spike could interact with, inter alia, bacteria.....
All I could think of was the Tetz researchers:
https://www.mdpi.com/2076-2607/6/2/54/htm
Spike enters bacteria?
Phages as new human viral pathogens a la the Tetz researchers?
"Here, for the first time, we presented the concept of bacteriophages as human pathogens, implying phages as previously overlooked factors that might be associated with human diseases. This concept adds phages and phagobiota to the growing list of factors associated with human health, and moreover suggests that bacteriophages and the alteration of their abundances may be a target for therapeutic intervention. Substantial additional experimental work is required to distinguish which bacteriophages contribute to the development of human diseases and to evaluate the roles of human’s genetic and microbiota susceptibilities therein."
Plus, this"https://lightonlight.education/ italian-researchers-vaccines-will-not-work-because-sars-cov-2-is-also-entering-bacteria
By the way, from March, There is the study, Hijacking time: How Ophiocordyceps fungi could be using ant host clocks to manipulate behavior. https://pubmed.ncbi.nlm.nih.gov/35103986/
Immunological Mechanisms Explaining the Role of Vaccines, IgE, Mast Cells, Histamine, Elevating Ferritin, IL-6, D-dimer, VEGF Levels in COVID-19 and Dengue, Potential Treatments Such as Mast Cell Stabilizers, Antihistamines: Predictions and Confirmations
https://europepmc.org/article/PPR/PPR241819
The mRNA vaccines are contaminated with endotoxins.
https://vinuarumugham.substack.com/p/leaked-eu-pfizer-agreement-reveals
This is insanity, from my next review - researchers actually used LPS to deliberately induce brain injury:
Wang X, Yu JY, Sun Y, Wang H, Shan H, Wang S. Baicalin protects LPS-induced blood-brain barrier damage and activates Nrf2-mediated antioxidant stress pathway. Int Immunopharmacol. 2021 Jul;96:107725. doi: 10.1016/j.intimp.2021.107725. Epub 2021 May 28. PMID: 34162131.
https://www.sciencedirect.com/science/article/pii/S1567576921003611
Dr Isaac Elias researches modified citrus pectin to lower galectin-3. A search of his name will bring up many interviews he has done on subject. His company EcoNugenics sell his version of modified citrus pectin called Pectasol. Just had my galectin levels drawn curious to see what they are had Covid in April.
Luc Montagnier said spike contains HIV sequences and a "malaria germ" and I keep wondering what that means...
I’ll look forward to it. I’ve also seen some preliminary data indicating that folic acid disrupts attachment of the spike protein to the furin binding site. So that could be additive to your discussion of natto.
I’m probably way off, but could any of this explain the “extremely unusual” autopsy findings in the congressman’s daughter’s (SADS victim) blood culture? Clostridium sordellii and septicum? Even if they didn’t consider it to be a contributing factor?