Regarding your comment on the vaccinated plasma: "One thing I will argue to death, and will mildly criticize the authors, is the “vaccinated plasma” and this mechanism not happening among vaccinated individuals. Perhaps vaccination timing and getting the plasma, perhaps methodology, or at a small chance, a different mechanism, but it is happening disproportionately among vaccinated individuals."
....why would they obfuscate with this statement?
What if this is true? The unvaccinated would be most at risk...right?
Authors didn’t test properly for this mechanism in vaccinated plasma, this is a fact. Why, maybe time, or grant (running these is expensive), maybe they would not get published in Nature if they said otherwise ?
If this only happens among unvaccinated, yes they would be most at risk, but just a very small percentage, and there are ways to stop it, either with specific monoclonals, or as I pointed out, dietary/supplement intervention (ketones/ketogenic diet/fasting).
One reader asked D. Sirotkin a question which he could not answer: "'I'm still waiting on the explanation for how convergent evolution happens in species thousands of miles away. I have yet to hear/read a convincing explanation of how this "works", and given your penchant for weaving substack tease-prose I'm confident you could spread the explanation out over months of posts keeping me interested the whole way." Answer: "And unfortunately I'm not gonna be able to answer that one, it definitely happens but how and why exactly isn't understood yet."
Back in December 2020-January 2021, I reminded everyone of the fact that HeLa cells were discovered back in the 1930s by Kurt Blome (a technology later imported to the US in the 1950s). Blome had discovered that bacteria communicate, over huge distances (thousands of km), through radio waves. Spike proteins = liquid crystals = T-bacilli = prions, as they also can use radio waves to send e/m copies of the cells from the vaccinated/infected people to the mycobacterium in the atmosphere (cometary dust which contains M. avium, M. influenzae, M. africanum). This is how the D614G strain replaced the Wuhan variant, back in march 2020, in just two weeks.
So why the Omicron (Mers-Cov-2) is not as virulent as the Delta variant? Because its PRION DOMAIN has not been activated. In contrast to the Delta variant (which has a powerful prion domain/region activated), Omicron does not feature a prion domain in the spike protein, it is latent. Now we know that people who develop IgA abs BUT NOT IgG do not get Covid-19. Covid-19 means the development of IgG abs. Prions are related to IgG abs. REGN10987 and B38 abs have a IgG format. Mers-Cov is related to IgG abs.
Therefore, only the NECESSARY condition for a real pandemic has been established, a VOC variant only. Now, we are waiting for the SUFFICIENT condition to appear on the scene, a VOHC variant (variant of high consequence). If the prion domain of the Omicron variant will be activated, it will cause a disaster.
Fascinating . I have long considered that astrology may involve a wave type vibratory implication from celestial bodies. If so, then wave type implications can also be contrived toward conditioning biology by intelligent design. If we consider either a multitude array of satellites or big instillations like HAARP it may be possible to target domains with wave forms that may condition pseudo progenitor type life forms , snd toward pathogenic outcomes.
I heard a podcast on agronomy where a scientist was specifically discussing his findings , but limited to what they can speak about because they were working for DOD . I believe it was a John Kempf regenerative agriculture podcast .
https://onlinelibrary.wiley.com/doi/10.1002/eji.202149655 (serum IgG-dominated responses correlated with T-cell responses to SARS-CoV-2 and PCR-confirmed Covid-19, whereas IgA-dominated responses correlated with not contracting the infection)
Severe Covid-19 cases are caused by two lethal antibodies: REGN10987 and B38, both of which have a IgG format.
On astrobiology:
"On October 11 2019 a meteoritic bolide (probably fragment of a comet) explodes in a brief flash in Nth East China. We think it probable that this bolide contained embedded within it a monoculture of infective nCoV-2019 virus particles that survived in the interior of the incandescent meteor.
Also well documented is that, in the winter of 1918, the disease appeared suddenly in the frozen wastes of Alaska, in villages that had been isolated for several months. Mathematical modelling of epidemics such as the one described invariably involves the ad hoc introduction of many unproven hypotheses—for example, that of the superspreader. In situations where proven infectivity is limited only to close contacts, a super-spreader is someone who can, on occasion, simultaneously infect a large number of susceptible individuals, thus causing the sporadic emergence of new clusters of disease. The recognition of a possible vertical input of external origin is conspicuously missing in such explanations.
With respect to the SARS outbreak, a prima facie case for a possible space incidence can already be made. First, the virus is unexpectedly novel, and appeared without warning in mainland China. A small amount of the culprit virus introduced into the stratosphere could make a first tentative fall out East of the great mountain range of the Himalayas, where the stratosphere is thinnest, followed by sporadic deposits in neighbouring areas. If the virus is only minimally infective, as it seems to be, the subsequent course of its global progress will depend on stratospheric transport and mixing, leading to a fall out continuing seasonally over a few years. Although all reasonable attempts to contain the infective spread of SARS should be continued, we should remain vigilant for the appearance of new foci (unconnected with infective contacts or with China) almost anywhere on the plant. New cases might continue to appear until the stratospheric supply of the causative agent becomes exhausted."
Let us remember that the huge wave in the fall of 1918 had arrived only after the huge eruption of the Katla volcano (12 October, 1918); the catalyst was the high/toxic doses of aspirin which had been administered to tens of millions of people, right now the catalyst is represented by the cmRNA/adenovirus vaccines.
Makes sense, I suppose, since the vaccine induces higher antibody titers than infection in most, or similar antibody titers to those who end up intensively ill or dead.
'One thing I will argue to death, and will mildly criticize the authors, is the “vaccinated plasma” and this mechanism not happening among vaccinated individuals. Perhaps vaccination timing and getting the plasma, perhaps methodology, or at a small chance, a different mechanism, but it is happening disproportionately among vaccinated individuals. It is literally the case I presented in the post below. '
Apologies I'm lost on this here... Could you re-write this for a bit more clarity?
Antibody dependent, well, anything, is happening in the vaccinated. I argue they used the wrong methods, and so they didn't found it, but it is happening. Might take months for someone to prove with research but it is. I will write more about it the next few days.
I am writing an article right now and I will link to yours, great job.
Eager to read it, and thank you for the complement =)
compl *I* ment.
I've gone back to normal life, and don't have anymore time for end-of-times speculation.
It's all about end-of-times preparedness, so that it's not end-of-times.
I see fake economies collapsing, real economies, in-the-end, striving.
Hi Igor, I went looking for your article, can you share the link? I did not see it in your list on Substack.
Regarding your comment on the vaccinated plasma: "One thing I will argue to death, and will mildly criticize the authors, is the “vaccinated plasma” and this mechanism not happening among vaccinated individuals. Perhaps vaccination timing and getting the plasma, perhaps methodology, or at a small chance, a different mechanism, but it is happening disproportionately among vaccinated individuals."
....why would they obfuscate with this statement?
What if this is true? The unvaccinated would be most at risk...right?
Authors didn’t test properly for this mechanism in vaccinated plasma, this is a fact. Why, maybe time, or grant (running these is expensive), maybe they would not get published in Nature if they said otherwise ?
If this only happens among unvaccinated, yes they would be most at risk, but just a very small percentage, and there are ways to stop it, either with specific monoclonals, or as I pointed out, dietary/supplement intervention (ketones/ketogenic diet/fasting).
One reader asked D. Sirotkin a question which he could not answer: "'I'm still waiting on the explanation for how convergent evolution happens in species thousands of miles away. I have yet to hear/read a convincing explanation of how this "works", and given your penchant for weaving substack tease-prose I'm confident you could spread the explanation out over months of posts keeping me interested the whole way." Answer: "And unfortunately I'm not gonna be able to answer that one, it definitely happens but how and why exactly isn't understood yet."
Back in December 2020-January 2021, I reminded everyone of the fact that HeLa cells were discovered back in the 1930s by Kurt Blome (a technology later imported to the US in the 1950s). Blome had discovered that bacteria communicate, over huge distances (thousands of km), through radio waves. Spike proteins = liquid crystals = T-bacilli = prions, as they also can use radio waves to send e/m copies of the cells from the vaccinated/infected people to the mycobacterium in the atmosphere (cometary dust which contains M. avium, M. influenzae, M. africanum). This is how the D614G strain replaced the Wuhan variant, back in march 2020, in just two weeks.
So why the Omicron (Mers-Cov-2) is not as virulent as the Delta variant? Because its PRION DOMAIN has not been activated. In contrast to the Delta variant (which has a powerful prion domain/region activated), Omicron does not feature a prion domain in the spike protein, it is latent. Now we know that people who develop IgA abs BUT NOT IgG do not get Covid-19. Covid-19 means the development of IgG abs. Prions are related to IgG abs. REGN10987 and B38 abs have a IgG format. Mers-Cov is related to IgG abs.
Therefore, only the NECESSARY condition for a real pandemic has been established, a VOC variant only. Now, we are waiting for the SUFFICIENT condition to appear on the scene, a VOHC variant (variant of high consequence). If the prion domain of the Omicron variant will be activated, it will cause a disaster.
Fascinating . I have long considered that astrology may involve a wave type vibratory implication from celestial bodies. If so, then wave type implications can also be contrived toward conditioning biology by intelligent design. If we consider either a multitude array of satellites or big instillations like HAARP it may be possible to target domains with wave forms that may condition pseudo progenitor type life forms , snd toward pathogenic outcomes.
I heard a podcast on agronomy where a scientist was specifically discussing his findings , but limited to what they can speak about because they were working for DOD . I believe it was a John Kempf regenerative agriculture podcast .
https://rense.com/general96/K-20220328/CREUTZFELDT-JACOB%20SARS-COV-2.pdf (Delta has a prionic domain, but not Omicron)
https://onlinelibrary.wiley.com/doi/10.1002/eji.202149655 (serum IgG-dominated responses correlated with T-cell responses to SARS-CoV-2 and PCR-confirmed Covid-19, whereas IgA-dominated responses correlated with not contracting the infection)
Severe Covid-19 cases are caused by two lethal antibodies: REGN10987 and B38, both of which have a IgG format.
On astrobiology:
"On October 11 2019 a meteoritic bolide (probably fragment of a comet) explodes in a brief flash in Nth East China. We think it probable that this bolide contained embedded within it a monoculture of infective nCoV-2019 virus particles that survived in the interior of the incandescent meteor.
Also well documented is that, in the winter of 1918, the disease appeared suddenly in the frozen wastes of Alaska, in villages that had been isolated for several months. Mathematical modelling of epidemics such as the one described invariably involves the ad hoc introduction of many unproven hypotheses—for example, that of the superspreader. In situations where proven infectivity is limited only to close contacts, a super-spreader is someone who can, on occasion, simultaneously infect a large number of susceptible individuals, thus causing the sporadic emergence of new clusters of disease. The recognition of a possible vertical input of external origin is conspicuously missing in such explanations.
With respect to the SARS outbreak, a prima facie case for a possible space incidence can already be made. First, the virus is unexpectedly novel, and appeared without warning in mainland China. A small amount of the culprit virus introduced into the stratosphere could make a first tentative fall out East of the great mountain range of the Himalayas, where the stratosphere is thinnest, followed by sporadic deposits in neighbouring areas. If the virus is only minimally infective, as it seems to be, the subsequent course of its global progress will depend on stratospheric transport and mixing, leading to a fall out continuing seasonally over a few years. Although all reasonable attempts to contain the infective spread of SARS should be continued, we should remain vigilant for the appearance of new foci (unconnected with infective contacts or with China) almost anywhere on the plant. New cases might continue to appear until the stratospheric supply of the causative agent becomes exhausted."
https://www.gjenvick.com/Influenza/IsTheInfluenzaAChinesePlague-1918-12.html (the only person who has ever found the true cause of the 1918 Spanish flu pandemic)
Let us remember that the huge wave in the fall of 1918 had arrived only after the huge eruption of the Katla volcano (12 October, 1918); the catalyst was the high/toxic doses of aspirin which had been administered to tens of millions of people, right now the catalyst is represented by the cmRNA/adenovirus vaccines.
Mycobacterium and radio waves:
https://arxiv.org/pdf/1501.01620.pdf
https://arxiv.org/pdf/1104.3113.pdf
Oh wow! So the vaccinated might be able to decrease viral replication BUT they will experience far more cell death in exchange.
Who doesn’t love 20% of your monocytes ending up in highly inflammatory cell death every infection. Taste like rainbow.
Makes sense, I suppose, since the vaccine induces higher antibody titers than infection in most, or similar antibody titers to those who end up intensively ill or dead.
https://pubmed.ncbi.nlm.nih.gov/22307489/
'One thing I will argue to death, and will mildly criticize the authors, is the “vaccinated plasma” and this mechanism not happening among vaccinated individuals. Perhaps vaccination timing and getting the plasma, perhaps methodology, or at a small chance, a different mechanism, but it is happening disproportionately among vaccinated individuals. It is literally the case I presented in the post below. '
Apologies I'm lost on this here... Could you re-write this for a bit more clarity?
Antibody dependent, well, anything, is happening in the vaccinated. I argue they used the wrong methods, and so they didn't found it, but it is happening. Might take months for someone to prove with research but it is. I will write more about it the next few days.