Unlike some of the other virus posts, this one will cover more than one paper. Second, and third, paper are good news.
Paper here.
Besides the abstract, this one will be a little bit harder to understand for the average Joe, but I will try to help you. I will point you towards some of my other posts later.
Researchers tested the cytokines and chemokines in patients with Multisystem Inflammatory Syndrome in Children, hyper inflammation in children after Covid, it can do a lot of damage, so knowing how to mitigate, or outright stop it, is a big deal, now that the virus infects children indiscriminately. And they suggest a pathway (wait for it.
To fully understand the impact of this paper you need to read the Reverse AIDS hypothesis, all parts preferably, but specially Part III, and the following. In Part IV, pay special attention to the IRIS part, if not, you won’t make sense of what I am about to propose.
To simplify, by testing the specific proteins in the blood of children with this specific inflammatory syndrome post-infection, they found specific cytokines were more present than others. Those were Interleukin-18 (just read my last post), Interleukin-6, and in another study (and in this one later) Interleukin-17A and Interferon Gamma.
I suggested this before, but will state it here again. In my opinion, MIS-C is just another form of IRIS, caused by a massive drop, and suppression of the child immune system, followed by its “reconstitution”, which sets off the inflammatory cascade. Safe to deduce, some of the children that go through MIS-C, will experience higher rates of autoimmunity, among other diseases, as years go by. Specially if vaccinated.
For the record, I think this will happen in a small percentage of the vaccinated.
“Wtf, why are you sharing some molecule that probably nobody has access ?”. Simple, we had a TMPRSS2 for quite a long while now, they just found new ones. With more uses than one, and given where it looks like the wind is shifting, better now, then never. This was first discovered in 2020 my someone I consider the best on his field.
Bromhexine/Ambroxol. It is a potent inhibitor to this precise mechanism, TMPRSS2, you can import in most countries in the world, including the FDA, even though it is prescription only in the US, under FDA rules you can import it, as long it is not for distribution, meaning, do NOT buy a dozen damn boxes.
The binding kinects are “hard coded” in the virus, every new variant had a somewhat stronger binding to TMPRSS2, so this will always help. Bromhexine is also one of the few things that can go into your lungs, specially in a pneumonia scenario. And the best thing of all.
It enhances the effect of antibiotics treatment, making the antibiotics penetrate biofilms.
Methylene Blue Is a Nonspecific Protein-Protein Interaction Inhibitor with Potential for Repurposing as an Antiviral for COVID-19
Pretty easy to understand =). PPI-Protein Protein Interaction inhibition is a very powerful tool to have in your ‘‘stack’’.
Thank you, just bought some methylene blue for my stash. 👍🏽
I looked at the full article on Methylene Blue - it talked about treatment after covid but was less specific about it as a preventative. Doses of 75 to 100 t.i.d. were mentioned as treatment doses. What is t.i.d.?