The case for Reverse Marek's

For months, we had strong signals which clearly showed the vaccines caused immune suppression and made any vaccinated person susceptible to infection. There are numerous problems with these vaccines, I will keep at the personal health level for now, because they do create all the turbocharged, highly adapted variants.

Chief Nerd @TheChiefNerd

UK Public Health data shows an average DECREASE of 7% in COVID-19 hospitalizations among the Unvaccinated, while those with 3+ doses saw an average INCREASE of 19% from last week These are rates per 100k by vaxxed and unvaxxed populations

Denmark data.

This preprint tells the history of a waning protection after a few months, the jab is useful for an amazing 50 days tops, at the cost of you getting the disease anyway and playing russian roulette with autoimmunity, cancer, and neurodegeneration.

I saw this paper (The inactivated NDV-HXP-S COVID-19 vaccine induces a significantly higher ratio of neutralizing to non-neutralizing antibodies in humans as compared to mRNA vaccines)

A paper forgotten to time. (The bold part is…quite…telling…)

The potential danger of suboptimal antibody responses in COVID-19

The quality and quantity of the antibody response dictates functional outcomes. High-affinity antibodies can elicit neutralization by recognizing specific viral epitopes (Fig.1a). Neutralizing antibodies are defined in vitro by their ability to block viral entry, fusion or egress.
In addition, neutralizing antibodies can interact with other immune components, including complement, phagocytes and natural killer cells. These effector responses can aid in pathogen clearance, with engagement of phagocytes shown to enhance antibody-mediated clearance of SARS-CoV4. However, in rare cases, pathogen-specific antibodies can promote pathology, resulting in a phenomenon known as antibody-dependent enhancement (ADE).

b | In antibody-dependent enhancement of infection, low quality, low quantity, non-neutralizing antibodies bind to virus particles through the Fab domains. Fc receptors (FcRs) expressed on monocytes or macrophages bind to Fc domains of antibodies and facilitate viral entry and infection. c | In antibody-mediated immune enhancement, low quality, low quantity, non-neutralizing antibodies bind to virus particles. Upon engagement by the Fc domains on antibodies, activating FcRs with ITAMs initiate signalling to upregulate pro-inflammatory cytokines and downregulate anti-inflammatory cytokines. Immune complexes and viral RNA in the endosomes can signal through Toll-like receptor 3 (TLR3), TLR7 and/or TLR8 to activate host cells, resulting in immunopathology.

We argue that ADE should be given full consideration in the safety evaluation of emerging candidate vaccines for SARS-CoV-2. In addition to vaccine approaches, monoclonal antibodies could be used to tackle this virus. Unlike vaccine-induced antibodies, monoclonal antibodies can be engineered with molecular precision. Safe and effective neutralizing antibodies could be produced on a mass-scale for delivery to populations across the world in the coming months.

(You should write those TLRs, they will be incredibly important later.)

So months ago, while deep into researching the Reverse AIDS, sometimes we came up with uncommon pathways, that required extensive interdisciplinary research to make sense, and once in a while I usually came up with insane ideas, they were not even hypothesis.

One of those was the Protein Interaction, after your body breaks down the spike, could pieces of it interact with the rest of your body/immune system and create toxic reactions ? No, this is insanity, it’s me being crazy again haha…ha…h….

Toxicity of spike fragments SARS-CoV-2 S protein for zebrafish: A tool to study its hazardous for human health?

SARS-CoV-2 Spike Protein Elicits Cell Signaling in Human Host Cells: Implications for Possible Consequences of COVID-19 Vaccines

The world is suffering from the coronavirus disease 2019 (COVID-19) pandemic caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). SARS-CoV-2 uses its spike protein to enter the host cells. Vaccines that introduce the spike protein into our body to elicit virus-neutralizing antibodies are currently being developed. In this article, we note that human host cells sensitively respond to the spike protein to elicit cell signaling. Thus, it is important to be aware that the spike protein produced by the new COVID-19 vaccines may also affect the host cells. We should monitor the long-term consequences of these vaccines carefully, especially when they are administered to otherwise healthy individuals. Further investigations on the effects of the SARS-CoV-2 spike protein on human cells and appropriate experimental animal models are warranted.

You see, the problem is not merely that the vaccines immuno suppress you, or turning you into a super-spreader, or even helping the landscape of highly adapted viral mutation. The real issue is that now we have a Reverse Marek’s scenario, where the vaccinated create supercharged, high transmissible viral swarms, which their compromised immune system can’t deal with, in turn infecting and creating a cascade in vaccinated individuals.

The soluble (meaning a free floating protein part of something) spike protein will mimic the entire disease. Different parts of the spike will elicit different responses and activate different cell signalling, which cascades into inflammatory responses. Hospitalizations lag, and we are clearing dealing with what I referred months ago as “hyper inflammatory cascade response”, which is just another way of saying ADE but with levels added to it.

If you want to start understanding this mess, here is a breadcrumb. It is tied with our entire hypothesis, which I might write or not, undecided. You can also tell why I named this the Everything Disease (Paper)