New virus found by WIV scientist in Kenya
Never ending journey !
Oh, the irony. A little time ago, the Chinese confirmed and released information about a new outbreak of a novel virus.
So before this goes viral, and since most ideologically driven people forget to do the research (let alone connect the dots) let’s delve into this novel virus, shall we ? Here is the paper published in the Chinese journal Virologica Sinica.
Emergence and re-emergence of infectious diseases of wildlife origin have led pre-emptive pathogen surveillances in animals to be a public health priority. Rodents and shrews are among the most numerically abundant vertebrate taxa and are known as natural hosts of important zoonotic viruses. Many surveillance programs focused more on RNA viruses. In comparison, much less is known about DNA viruses harbored by these small mammals. To fill this knowledge gap, tissue specimens of 232 animals including 226 rodents, five shrews and one hedgehog were collected from 5 counties in Kenya and tested for the presence of DNA viruses belonging to 7 viral families by PCR. Diverse DNA sequences of adenoviruses, adeno-associated viruses, herpesviruses and polyomaviruses were detected. Phylogenetic analyses revealed that most of these viruses showed distinction from previously described viruses and formed new clusters. Furthermore, this is the first report of the discovery and full-length genome characterization of a polyomavirus in Lemniscomys species. This novel polyomavirus, named LsPyV KY187, has less than 60% amino acid sequence identity to the most related Glis glis polyomavirus 1 and Sciurus carolinensis polyomavirus 1 in both large and small T-antigen proteins and thus can be putatively allocated to a novel species within Betapolyomavirus. Our findings help us better understand the genetic diversity of DNA viruses in rodent and shrew populations in Kenya and provide new insights into the evolution of those DNA viruses in their small mammal reservoirs. It demonstrates the necessity of ongoing pathogen discovery studies targeting rodent-borne viruses in East Africa.
Per the paper, they belong to one of the many surveillance programs, but unlike most focused on RNA viruses, this one is focused on DNA viruses. They found a few new ones but the one that really got their attention was the new polyomavirus. This novel polyomavirus was so distinct from others from the same “family” that the authors advise creating its own distinct classification. Nothing much else about it besides genetics and classification and evolutionary discussion on this virus.
Also, there hasn’t been a human infection with this particular virus as far as we know it, unlike the Langya virus which we have dozens of official cases and most likely a couple more dozens that went under the radar.
But polyomavirus led me on an intriguing road.
Polyomaviruses and human diseases
Progressive multifocal leukoencephalopathy is a demyelinating CNS disorder. Reactivation of John Cunningham virus leads to oligodendrocyte infection with lysis and consequent axonal loss due to demyelination. Patients usually present with confusion and seizures
What I am getting at ? That there are dynamics at play that perhaps a sizable portion of clinicians are overlooking, in regards to what the Spike Protein is doing in some people.
Progressive multifocal leukoencephalopathy (PML) is a rare but fatal disease leading to severe neurological impairments. PML is a clinical manifestation, which is usually associated with John Cunningham virus (JCV) infection. It is also correlated to malignancies that mainly include hematologic malignancies such as chronic lymphocytic leukemia (CLL). Until now, no specific treatment has been established for JCV-induced PML; therefore, the prognosis of this disease is poor.We present a case of a 67-year-old woman who suffered from CLL with a chief complaint of seizure. Her clinical symptoms, results of brain MRI and biopsy were suggestive for the JCV-induced PML. The patient had received treatment using mefloquine at dose of 250 mg/day with no clinical improvement.
From a friend.
Community mourns CA high school football player who died during surgery. ‘Special kid’
It was later discovered that he had a heart tumor related to an undiagnosed T-cell leukemia
Rest in peace kid, lately a few cases of “rare blood disorder” have been hitting the news cycle, and no I don’t think the average person should be worried, what I am getting at is the following.
If there is a correlation between the Spike damage HSPCs, the immune suppressive state a portion of the population definitely finds itself in every so often, and untested/undetected progress of latent infections mentioned above. No, I don’t think this is a massive problem, no you should not be worried, especially if you take care of your health, just bringing some points because I know there is a myriad of different people from all sorts of fields that read this. These all share receptors that SARS-CoV-2 messes with… and so do the vaccines.
How many new diseases will this pandemic turbo charge into rapid evolution ?
This is also how my brain works.
Massive appreciation to all supporters here and on Kofi !!!
Don't want to make a post laughing at this, but there was a meme in 2020 of SARS-CoV-2 spreading and infecting from a dead body, we had a good time, and laughs with that one.
Well... turns out it is kinda true LOL -> https://www.biorxiv.org/content/10.1101/2022.08.29.505777v1
Please Read Walter M. Chestnut on Substack to understand the dangers of the spike protein.